Hi Sara I am carlo, regarding steroids I was always taking depo cyp testosterone 10ml week , sum tyms stop taking for 4 weeks,on an of ,sex driver was always gud,, but after dat I quit for gud for week, but as I am gyming I started taking testo blast zma Sa supplements an creatine hcl e amino acid 6000, after3 days taking dos supplements I am experiencing Low libido, but wen I play with my cook dase erect but tek long,even I stopped taking dos supplements for 4 days stl having sem problem . Must I get hcg to kick start natural testerone?
Hey I’m 18 and have been lifting since I was in 7th grade and am now a senior. I’m interested In the dball cycle over the dianabol but have a few questions. The first question is do I need to take a test booster with it? Although it is recommended Ik if you mess with your body’s natural production at a young age it can screw up your production of it. My next question is about after you finish it. I’m seeing stuff about if the effects last and what I’m asking is if the dball effects of muscle growth wear off or do you lose the muscle you gained. What iv got from reading is if you just take the pills daily without test booster which is my option I’m really wanting to take, after my cycle runs out I won’t just lose muscle or stop growth will I? Thanks for you’re time and get back to me asap as I’m looking to order it soon
A 31-year-old man presenting with an 18-month history of sexual dysfunction resulting from severe adult-onset IHH (LH U/L, FSH U/L, T nmol/L). Initial therapy with 50 mg of clomiphene citrate (CC) three times a day for 7 days, with overnight LH pulse profiling and 9 am T levels evaluated at baseline and on completion. A 2-month washout period, followed by low-dose maintenance therapy (25-50 mg/d) for 4 months.
MAIN OUTCOME MEASURE(S):Baseline and stimulated T levels and LH pulsatility; effect on sexual function.
RESULT(S):Clomiphene therapy resulted in complete normalization of pulsatile gonadotropin secretion, serum T level, and sexual function. CONCLUSION(S):Isolated hypogonadotropic hypogonadism may result from an acquired defect of enhanced hypothalamic sensitivity to E-mediated negative feedback. Whereas direct T replacement therapy can further suppress endogenous gonadotropin secretion, treating IHH men with gonadotropins can stimulate endogenous T secretion and enhance fertility potential. On theoretical grounds, reversal of gonadotropin deficiency with CC might be expected to have a similar biological effect.